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I’m feeling run down. Why am I more likely to get sick? And how can I boost my immune system?

<div class="theconversation-article-body"> <a href="https://theconversation.com/profiles/sathana-dushyanthen-1169328">Sathana Dushyanthen</a>, <a href="https://theconversation.com/institutions/the-university-of-melbourne-722">The University of Melbourne</a> It has been a long winter, filled with many viruses and cost-of-living pressures, on top of the usual mix of work, study, life admin and caring responsibilities. Stress is an inevitable part of life. In short bursts, our stress response has evolved as a survival mechanism to help us be more alert in <a href="https://theconversation.com/no-you-cant-blame-all-your-health-issues-on-high-cortisol-heres-how-the-hormone-works-203162">fight or flight situations</a>. But when stress is chronic, it weakens the immune system and makes us more vulnerable to illnesses such as the <a href="https://www.healthline.com/health/can-stress-make-you-sick#:%7E:text=The%20common%20cold&text=Inflammation%20has%20been%20linked%20to,to%20the%20cold%2Dcausing%20germs.">common cold</a>, <a href="https://journals.lww.com/psychosomaticmedicine/abstract/1999/03000/psychological_stress,_cytokine_production,_and.9.aspx">flu</a> and <a href="https://onlinelibrary.wiley.com/doi/abs/10.1002/smi.3017">COVID</a>. <h2>Stress makes it harder to fight off viruses</h2> When the immune system starts to break down, a virus that would normally have been under control starts to flourish. Once you begin to feel sick, the <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4465119/">stress response</a> rises, making it harder for the immune system to fight off the disease. You may be sick more often and for longer periods of time, without enough immune cells primed and ready to <a href="https://link.springer.com/chapter/10.1007/978-3-030-16996-1_6">fight</a>. In the 1990s, American psychology professor Sheldon Cohen and his colleagues conducted a number of <a href="https://www.cmu.edu/common-cold-project/">studies</a> where healthy people were exposed to an upper respiratory infection, through drops of virus placed directly into their <a href="https://www.nejm.org/doi/full/10.1056/NEJM199108293250903">nose</a>. These participants were then quarantined in a hotel and monitored closely to determine who became <a href="https://theconversation.com/stress-less-it-might-protect-you-from-covid-153361">ill</a>. One of the most important factors predicting who got sick was prolonged psychological <a href="https://journals.sagepub.com/doi/full/10.1177/1745691620942516">stress</a>. <h2>Cortisol suppresses immunity</h2> “Short-term stress” is stress that lasts for a period of minutes to hours, while “chronic stress” persists for several hours per day for <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5964013/#:%7E:text=Therefore%2C%20a%20major%20distinguishing%20characteristic,weeks%20or%20months%20%5B9%5D.">weeks or months</a>. When faced with a perceived threat, psychological or physical, the hypothalamus region of the brain sets off an alarm system. This signals the release of a surge of hormones, including adrenaline and <a href="https://www.embopress.org/doi/full/10.15252/msb.20209510">cortisol</a>. <figure class="align-center "><img src="https://images.theconversation.com/files/616892/original/file-20240903-18-blrqoz.png?ixlib=rb-4.1.0&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/616892/original/file-20240903-18-blrqoz.png?ixlib=rb-4.1.0&q=45&auto=format&w=600&h=472&fit=crop&dpr=1 600w, https://images.theconversation.com/files/616892/original/file-20240903-18-blrqoz.png?ixlib=rb-4.1.0&q=30&auto=format&w=600&h=472&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/616892/original/file-20240903-18-blrqoz.png?ixlib=rb-4.1.0&q=15&auto=format&w=600&h=472&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/616892/original/file-20240903-18-blrqoz.png?ixlib=rb-4.1.0&q=45&auto=format&w=754&h=593&fit=crop&dpr=1 754w, https://images.theconversation.com/files/616892/original/file-20240903-18-blrqoz.png?ixlib=rb-4.1.0&q=30&auto=format&w=754&h=593&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/616892/original/file-20240903-18-blrqoz.png?ixlib=rb-4.1.0&q=15&auto=format&w=754&h=593&fit=crop&dpr=3 2262w" alt="Human brain illustration" /><figcaption>The hypothalamus sets off an alarm system in response to a real or perceived threat. <a class="source" href="https://www.shutterstock.com/image-illustration/hypothalamus-causes-vasoconstriction-illustration-medical-brain-435142264">stefan3andrei/Shutterstock</a></figcaption></figure> In a typical stress response, <a href="https://www.sciencedirect.com/science/article/abs/pii/S147149060300173X">cortisol levels</a> quickly increase when stress occurs, and then rapidly drop back to normal once the stress has subsided. In the short term, cortisol suppresses inflammation, to ensure the body has enough energy available to respond to an <a href="https://www.frontiersin.org/journals/immunology/articles/10.3389/fimmu.2019.00245/full">immediate threat</a>. But in the longer term, chronic stress can be harmful. A Harvard University study <a href="https://jamanetwork.com/journals/jamapsychiatry/fullarticle/2796097">from 2022</a> showed that people suffering from psychological distress in the lead up to their COVID infection had a greater chance of experiencing long COVID. They <a href="https://jamanetwork.com/journals/jamapsychiatry/fullarticle/2796097">classified</a> this distress as depression, probable anxiety, perceived stress, worry about COVID and loneliness. Those suffering distress had close to a <a href="https://jamanetwork.com/journals/jamapsychiatry/fullarticle/2796097">50% greater risk</a> of long COVID compared to other <a href="https://theconversation.com/being-stressed-out-before-you-get-covid-increases-your-chances-of-long-covid-heres-why-190649">participants</a>. Cortisol has been shown to be high in the most severe cases of <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7102614/">COVID</a>. <h2>Stress causes inflammation</h2> <a href="https://stories.uq.edu.au/imb/the-edge/inflammation/what-is-inflammation/index.html">Inflammation</a> is a short-term reaction to an injury or infection. It is responsible for trafficking immune cells in your body so the right cells are present in the right locations at the right times and at the right <a href="https://link.springer.com/article/10.1007/s12026-014-8517-0">levels</a>. The immune cells also store a memory of that threat to respond faster and more effectively the next <a href="https://theconversation.com/being-stressed-out-before-you-get-covid-increases-your-chances-of-long-covid-heres-why-190649">time</a>. Initially, circulating immune cells detect and flock to the site of <a href="https://www.nature.com/articles/ni1275">infection</a>. Messenger proteins, known as pro-inflammatory cytokines, are released by immune cells, to signal the danger and recruit help, and our immune system responds to neutralise the <a href="https://stories.uq.edu.au/imb/the-edge/inflammation/what-is-inflammation/index.html">threat</a>. During this response to the infection, if the immune system produces too much of these inflammatory chemicals, it can trigger symptoms such as nasal congestion and runny <a href="https://journals.sagepub.com/doi/full/10.1177/1745691620942516">nose</a>. <h2>What about chronic stress?</h2> Chronic stress causes persistently high cortisol secretion, which remains high even in the absence of an immediate <a href="https://theconversation.com/no-you-cant-blame-all-your-health-issues-on-high-cortisol-heres-how-the-hormone-works-203162">stressor</a>. The immune system becomes desensitised and unresponsive to this <a href="https://www.sciencedirect.com/science/article/abs/pii/S1043661816307435">cortisol suppression</a>, increasing low-grade “silent” inflammation and the production of pro-inflammatory cytokines (the messenger proteins). Immune cells become exhausted and start to <a href="https://www.fxmedicine.com.au/blog-post/adrenal-immune-connection">malfunction</a>. The body loses the ability to turn down the inflammatory <a href="https://journals.sagepub.com/doi/full/10.1177/1745691620942516">response</a>. Over time, the immune system changes the way it responds by reprogramming to a “<a href="https://www.unimelb.edu.au/newsroom/news/2021/april/how-stress-can-stop-immune-cells-in-their-tracks">low surveillance mode</a>”. The immune system misses early opportunities to destroy threats, and the process of recovery can take <a href="https://theconversation.com/being-stressed-out-before-you-get-covid-increases-your-chances-of-long-covid-heres-why-190649">longer</a>. <h2>So how can you manage your stress?</h2> We can actively strengthen our immunity and natural defences by managing our <a href="https://theconversation.com/stuck-in-fight-or-flight-mode-5-ways-to-complete-the-stress-cycle-and-avoid-burnout-or-depression-218599">stress levels</a>. Rather than letting stress build up, try to address it early and frequently by: 1) Getting enough sleep Getting enough sleep reduces <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3132857/">cortisol levels</a> and inflammation. During sleep, the immune system <a href="https://link.springer.com/chapter/10.1007/978-1-4939-6578-6_12">releases</a> <a href="https://theconversation.com/sleep-wont-cure-the-coronavirus-but-it-can-help-our-bodies-fight-it-134674">cytokines</a>, which help fight infections and inflammation. 2) Taking regular exercise Exercising helps the lymphatic system (which balances bodily fluids as part of the immune system) circulate and allows immune cells to monitor for threats, while sweating flushes <a href="https://www.healthline.com/nutrition/does-exercise-boost-immune-system">toxins</a>. Physical activity also lowers stress hormone levels through the release of positive brain <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7387807/">signals</a>. 3) Eating a healthy diet Ensuring your diet contains enough nutrients – such as the B vitamins, and the full breadth of minerals like magnesium, iron and zinc – during times of stress has a positive impact on overall stress <a href="https://www.ncbi.nlm.nih.gov/pubmed/22782571">levels</a>. Staying hydrated helps the body to flush out <a href="https://theconversation.com/a-strong-immune-system-helps-ward-off-colds-and-flus-but-its-not-the-only-factor-99512">toxins</a>. 4) Socialising and practising meditation or mindfulness These activities increase endorphins and serotonin, which improve mood and have <a href="https://www.sciencedirect.com/science/article/pii/S2949834123000351">anti-inflammatory effects</a>. Breathing exercises and meditation stimulate the parasympathetic nervous system, which calms down our stress responses so we can “reset” and reduce <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4940234/">cortisol levels</a>.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/237456/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/sathana-dushyanthen-1169328">Sathana Dushyanthen</a>, Academic Specialist & Lecturer in Cancer Sciences & Digital Health| Superstar of STEM| Science Communicator, <a href="https://theconversation.com/institutions/the-university-of-melbourne-722">The University of Melbourne</a> Image credits: Shutterstock This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/im-feeling-run-down-why-am-i-more-likely-to-get-sick-and-how-can-i-boost-my-immune-system-237456">original article</a>. </div>

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How long does immunity last after a COVID infection?

<a href="https://theconversation.com/profiles/lara-herrero-1166059">Lara Herrero</a>, <a href="https://theconversation.com/institutions/griffith-university-828">Griffith University</a> and <a href="https://theconversation.com/profiles/dr-wesley-freppel-1408971">Dr Wesley Freppel</a>, <a href="https://theconversation.com/institutions/griffith-university-828">Griffith University</a> Nearly four years into the pandemic, Australia, like many other countries, is still seeing large numbers of <a href="https://nindss.health.gov.au/pbi-dashboard/">COVID cases</a>. Some 860,221 infections were recorded around the country in 2023, while 30,283 cases have already been reported in 2024. This is likely to be a significant underestimate, with fewer people testing and reporting than earlier in the pandemic. But the signs suggest parts of Australia are experiencing yet <a href="https://www.abc.net.au/news/2024-01-23/covid-19-case-numbers-from-australia-states-and-territories/103374656">another COVID surge</a>. While some lucky people claim to have never had COVID, many are facing our second, third or even fourth infection, often despite having been vaccinated. You might be wondering, how long does immunity last after a previous infection or vaccination? Let’s take a look at what the evidence shows. <h2>B cells and T cells</h2> To answer this question, we need to understand a bit about how <a href="https://theconversation.com/what-happens-in-our-body-when-we-encounter-and-fight-off-a-virus-like-the-flu-sars-cov-2-or-rsv-207023">immunity</a> to SARS-CoV-2 (the virus that causes COVID) works. After being infected or vaccinated, the immune system develops specific antibodies that can neutralise SARS-CoV-2. B cells remember the virus for a period of time. In addition, the immune system produces memory T cells that can kill the virus, and remain in the blood for some months after the clearance of the infection or a vaccination. A <a href="https://www.science.org/doi/full/10.1126/science.abf4063?rfr_dat=cr_pub++0pubmed&url_ver=Z39.88-2003&rfr_id=ori%3Arid%3Acrossref.org">2021 study</a> found 98% of people had antibodies against SARS-CoV-2’s spike protein (a protein on the surface of the virus that allows it to attach to our cells) one month after symptom onset. Six to eight months afterwards, 90% of participants still had these neutralising antibodies in their blood. This means the immune system should have recognised and neutralised the same SARS-CoV-2 variant if challenged within six to eight months (if an infection occurred, it should have resulted in mild to no symptoms). <h2>But what about when the virus mutates?</h2> As we know, SARS-CoV-2 has mutated over time, leading to the emergence of new variants such as alpha, beta, delta and omicron. Each of these variants carries mutations that are new to the immune system, even if the person has been previously infected with an earlier variant. A new variant likely won’t be <a href="https://www.science.org/doi/10.1126/science.adj0070">perfectly recognised</a> – or even <a href="https://www.cell.com/cell/pdf/S0092-8674(21)01578-6.pdf">recognised at all</a> – by the already activated memory T or B cells from a previous SARS-CoV-2 infection. This could explain why people can be so readily reinfected with COVID. A recent <a href="https://www.thelancet.com/article/S0140-6736(22)02465-5/fulltext#seccestitle10">review of studies</a> published up to the end of September 2022 looked at the protection conferred by previous SARS-CoV-2 infections. The authors found a previous infection provided protective immunity against reinfection with the ancestral, alpha, beta and delta variants of 85.2% at four weeks. Protection against reinfection with these variants remained high (78.6%) at 40 weeks, or just over nine months, after the previous infection. This protection decreased to 55.5% at 80 weeks (18 months), but the authors noted there was a lack of data at this time point. Notably, an earlier infection provided only 36.1% protection against a reinfection with omicron BA.1 at 40 weeks. Omicron has been described as an <a href="https://www.nature.com/articles/s41564-022-01143-7">immune escape variant</a>. A prior infection showed a high level of protection against severe disease (above 88%) up to 40 weeks regardless of the variant a person was reinfected with. <h2>What about immunity after vaccination?</h2> So far almost 70 million COVID vaccines <a href="https://www.health.gov.au/topics/covid-19/reporting">have been administered</a> to more than <a href="https://www.health.gov.au/resources/publications/covid-19-vaccine-rollout-update-12-january-2023?language=en">22 million people</a> in Australia. Scientists estimated COVID vaccines prevented around <a href="https://www.thelancet.com/journals/laninf/article/PIIS1473-3099(22)00320-6/fulltext">14.4 million deaths</a> in 185 countries in the first year after they became available. But we know COVID vaccine effectiveness wanes over time. A <a href="https://jamanetwork.com/journals/jamanetworkopen/fullarticle/2804451?utm_source=For_The_Media&utm_medium=referral&utm_campaign=ftm_links&utm_term=050323">2023 review</a> found the original vaccines were 79.6% and 49.7% effective at protecting against symptomatic delta infection at one and nine months after vaccination respectively. They were 60.4% and 13.3% effective against symptomatic omicron at the same time points. This is where booster doses come into the picture. They’re important to keep the immune system ready to fight off the virus, particularly for those who are more vulnerable to the effects of a COVID infection. Plus, regular booster doses can provide immunity against different variants. COVID vaccines are constantly being <a href="https://mvec.mcri.edu.au/references/covid-19/">reviewed and updated</a> to ensure optimal protection against <a href="https://www.who.int/activities/tracking-SARS-CoV-2-variants">current circulating strains</a>, with the latest shot available designed to target <a href="https://www.health.gov.au/ministers/the-hon-mark-butler-mp/media/new-covid-19-vaccines-available-to-target-current-variants">the omicron variant XBB 1.5</a>. This is similar to how we approach seasonal flu vaccines. A <a href="https://www.nature.com/articles/s41598-023-50335-6">recent study</a> showed a COVID vaccination provides longer protection against reinfection than natural protection alone. The median time from infection to reinfection in non-vaccinated people was only six months, compared with 14 months in people who had received one, two or three doses of vaccine after their first infection. This is called <a href="https://www.science.org/doi/10.1126/science.abj2258">hybrid immunity</a>, and other research has similarly found it provides better protection than natural infection alone. It also seems timing is important, as receiving a vaccine too soon after an infection (less than six months) appears to be <a href="https://www.nature.com/articles/s41598-023-50335-6">less effective</a> than getting vaccinated later. <h2>What now?</h2> Everyone’s immune system is slightly unique, and SARS-CoV-2 continues to mutate, so knowing exactly how long COVID immunity lasts is complicated. Evidence suggests immunity following infection should generally last six months in healthy adults, and can be prolonged with vaccination. But there are exceptions, and all of this assumes the virus has not mutated so much that it “escapes” our immune response. While many people feel the COVID pandemic is over, it’s important we don’t forget the lessons we have learned. Practices such as wearing a mask and staying home when unwell can reduce the spread of many viruses, not only <a href="https://www.bmj.com/content/375/bmj-2021-068302">COVID</a>. Vaccination is not mandatory, but for older adults eligible for a booster under the <a href="https://www.health.gov.au/news/atagi-update-on-the-covid-19-vaccination-program">current guidelines</a>, it’s a very good idea.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/221398/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/lara-herrero-1166059">Lara Herrero</a>, Research Leader in Virology and Infectious Disease, <a href="https://theconversation.com/institutions/griffith-university-828">Griffith University</a> and <a href="https://theconversation.com/profiles/dr-wesley-freppel-1408971">Dr Wesley Freppel</a>, Research Fellow, Institute for Glycomics, <a href="https://theconversation.com/institutions/griffith-university-828">Griffith University</a> Image credits: Getty Images This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/how-long-does-immunity-last-after-a-covid-infection-221398">original article</a>.

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How biological differences between men and women alter immune responses – and affect women’s health

<a href="https://theconversation.com/profiles/helen-mcgettrick-1451122">Helen McGettrick</a>, <a href="https://theconversation.com/institutions/university-of-birmingham-1138">University of Birmingham</a> and <a href="https://theconversation.com/profiles/asif-iqbal-1451123">Asif Iqbal</a>, <a href="https://theconversation.com/institutions/university-of-birmingham-1138">University of Birmingham</a> Most people will have heard the term “man flu”, which refers to men’s perceived tendency to exaggerate the severity of a cold or a similar minor ailment. What most people may not know is that, generally speaking, women mount stronger <a href="https://pubmed.ncbi.nlm.nih.gov/36121220/">immune responses</a> to infections than men. Men are <a href="https://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1005374">more susceptible</a> to infections from, for example, HIV, hepatitis B, and Plasmodium falciparum (the parasite responsible for malaria). They can also have more severe symptoms, with evidence showing they’re more likely to be <a href="https://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1005374">admitted to hospital</a> when infected with hepatitis B, tuberculosis, and Campylobacter jejuni (a bacteria that causes gastroenteritis), among others. While this may be positive for women in some respects, it also means women are at <a href="https://www.nature.com/articles/nri2815">greater risk</a> of developing chronic diseases driven by the immune system, known as immune-mediated inflammatory diseases. Here we will explore how biological factors influence immune differences between the sexes and how this affects women’s health. While we acknowledge that both sex and gender may affect immune responses, this article will focus on biological sex rather than gender. <h2>Battle of the sexes</h2> There are differences <a href="https://www.nature.com/articles/nri.2016.90">between the sexes</a> at every stage of the immune response, from the number of immune cells, to their degree of activation (how ready they are to respond to a challenge), and beyond. However, the story is more complicated than that. Our immune system evolves throughout our lives, learning from past experiences, but also responding to the physiological challenges of getting older. As a result, <a href="https://www.nature.com/articles/nri.2016.90">sex differences</a> in the immune system can be seen from birth through puberty into adulthood and <a href="https://academic.oup.com/jleukbio/advance-article/doi/10.1093/jleuko/qiad053/7190870">old age</a>. Why do these differences occur? The first part of answering this question involves the X chromosome. Females have two X chromosomes, while males have one X and one Y chromosome. The <a href="https://pubmed.ncbi.nlm.nih.gov/20651746/">X chromosome</a> contains the largest number of immune-related genes. The X chromosome also has <a href="https://link.springer.com/article/10.1007/s00018-020-03526-7">around 118 genes</a> from a gene family that are able to stop the expression of other genes, or change how proteins are made, including those required for immunity. These gene-protein regulators are known as microRNA, and there are only <a href="https://pubmed.ncbi.nlm.nih.gov/24808907/">two microRNA genes</a> on the Y chromosome. The X chromosome has <a href="https://www.genome.gov/about-genomics/fact-sheets/X-Chromosome-facts">more genes overall</a> (around 900) than the Y chromosome (around 55), so female cells have evolved to switch off one of their X chromosomes. This is not like turning off a light switch, but more like using a dimmer. Around <a href="https://bmcgenomics.biomedcentral.com/articles/10.1186/s12864-019-5507-6">15-25% of genes</a> on the silenced X chromosome are expressed at any given moment in any given cell. This means female cells can often express more immune-related genes and gene-protein regulators than males. This generally means a faster clearance of pathogens in females than males. Second, men and women have <a href="https://www.frontiersin.org/articles/10.3389/fimmu.2020.604000/full">varying levels</a> of different sex hormones. Progesterone and testosterone are broadly considered to limit immune responses. While both hormones are produced by males and females, progesterone is found at higher concentrations in non-menopausal women than men, and testosterone is much higher in men than women. The role of <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6533072/">oestrogen</a>, one of the main female sex hormones, is more complicated. Although generally oestrogen <a href="https://www.sciencedirect.com/science/article/abs/pii/S000887491500026X?via%3Dihub">enhances immune responses</a>, its levels vary during the menstrual cycle, are high in pregnancy and low after menopause. Due in part to these genetic and hormonal factors, pregnancy and the years following are associated with heightened immune responses to external challenges such as infection. This has been regarded as an <a href="https://www.nature.com/articles/nri.2016.90">evolutionary feature</a>, protecting women and their unborn children during pregnancy and enhancing the mother’s survival throughout the child-rearing years, ultimately ensuring the survival of the population. We also see this pattern in <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2628977/">other species</a> including insects, lizards, birds and mammals. <h2>What does this all mean?</h2> With women’s heightened immune responses to infections comes an increased risk of certain diseases and prolonged immune responses after infections. An <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3328995/">estimated 75-80%</a> of all immune-mediated inflammatory diseases <a href="https://pubmed.ncbi.nlm.nih.gov/32542149/">occur in females</a>. Diseases more common in women include multiple sclerosis, <a href="https://www.nature.com/articles/nri2815">rheumatoid arthritis</a>, lupus, Sjogren’s syndrome, and <a href="https://www.nature.com/articles/nri.2016.90">thyroid disorders</a> such as Graves disease. In these diseases, the immune system is continuously fighting against what it sees as a foreign agent. However, often this perceived threat is not a foreign agent, but cells or tissues from the host. This leads to tissue damage, pain and immobility. Women are also prone to chronic inflammation following infection. For example, after infections with <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5818468/">Epstein Barr virus</a> or <a href="https://www.liebertpub.com/doi/10.1089/jwh.2008.1193">Lyme disease</a>, they may go on to develop <a href="https://www.nhs.uk/conditions/chronic-fatigue-syndrome-cfs/">chronic fatigue syndrome</a>, another condition that affects more women than men. This is one possible explanation for the heightened risk among <a href="https://www.frontiersin.org/articles/10.3389/fresc.2023.1122673/full">pre-menopausal women</a> of developing long COVID following infection with SARS-CoV-2, the virus that causes COVID. Research has also revealed the presence of auto-antibodies (antibodies that attack the host) in patients with long COVID, suggesting it might be an <a href="https://www.sciencedirect.com/science/article/pii/S1568997221000550">autoimmune disease</a>. As women are more susceptible to autoimmune conditions, this could potentially explain the sex bias seen. However, the exact causes of long COVID, and the reason women may be at greater risk, are yet to be defined. This paints a bleak picture, but it’s not all bad news. Women typically mount <a href="https://pubmed.ncbi.nlm.nih.gov/24966191/">better vaccine responses</a> to several common infections (for example, influenza, measles, mumps, rubella, hepatitis A and B), producing higher antibody levels than men. One study showed that women vaccinated with half a dose of flu vaccine produced the same amount of antibodies compared to men vaccinated with <a href="https://jamanetwork.com/journals/jamainternalmedicine/fullarticle/773453">a full dose</a>. However, these responses <a href="https://www.nature.com/articles/nri.2016.90">decline as women age</a>, and particularly <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3954964/">after menopause</a>. All of this shows it’s vital to consider sex when designing studies examining the immune system and treating patients with immune-related diseases.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/208802/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/helen-mcgettrick-1451122">Helen McGettrick</a>, Reader in Inflammation and Vascular Biology, <a href="https://theconversation.com/institutions/university-of-birmingham-1138">University of Birmingham</a> and <a href="https://theconversation.com/profiles/asif-iqbal-1451123">Asif Iqbal</a>, Associate Professor in Inflammation Biology, <a href="https://theconversation.com/institutions/university-of-birmingham-1138">University of Birmingham</a> Image credits: Getty Images This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/how-biological-differences-between-men-and-women-alter-immune-responses-and-affect-womens-health-208802">original article</a>.

Body

Outrage after renters confronted with cleaning “scoring system”

An Australian real estate agent has come under fire for their “demoralising” scoring system for tenants during routine inspections. One fired up renter said they were shocked to receive a scorecard from the agent after they complied with an inspection, and shared their feedback on Reddit. The scorecard rated the tenant’s efforts cleaning the property in 17 different rooms and areas, marking them on cleanliness, and if the areas were damaged or required maintenance. The renter was then also given an overall rating out of five stars, based on the upkeep of their rental home. The Reddit user showed that they received just three and half stars, despite being marked down in only one area due to a light globe not working. In their post, the tenant said they had made every effort to clean the home, adding that the rating was “demoralising”. “I’ve been renting a long time, and my deep cleaning routine is based around inspections,” they explained. “I go all out - every room in my little 3x2 villa is given a spring clean, every surface is washed including walls, doors, lights, windows, carpets etc.” “Had my latest inspection a few weeks ago and just received the report. Despite the agent commenting once again that she never sees houses as clean as mine, and taking 112 (!) photos of every single thing that shows how great the condition of the house is - having one light globe not working is so terrible that I’m scored three and a half out of five.” The renter went on to say that they had only recently discovered that the broken light globe was the real estate’s responsibility to replace, and does not warrant such a severe marking down in their “score”. “The globe in question is a downlight fitting. We have so many downlights that I don’t even use this particular one,” they continued. “I can’t replace the globe itself, I need to get into the roof to replace the whole unit. Thanks to people here I’ve learned this isn’t even my responsibility to fix!” The renter’s post sparked a huge outrage online, with many up in arms over the current state of the rental market and unrealistic expectations of estate agents. One person said, “I fully believe that the average renter keeps a home to a higher standard than an owner, and it seems deliberately demoralising that someone could be marked down for having a light out.” Another added, “This is ridiculously petty, and more importantly, totally irrelevant to the purpose of a periodic inspection. Which is to identify whether any maintenance is needed.” A third person took their anger out on the entire system, summing it up by writing, “I sort of wonder what little lightbulb clicks in your mind and you wake up one day and ask yourself, ‘How can I be a massive c*** who offers no value to society? I know! I will get into real estate.’” Image credits: Getty Images / Reddit

Real Estate

Why does my back get so sore when I’m sick? The connection between immunity and pain

<a href="https://theconversation.com/profiles/joshua-pate-1399299">Joshua Pate</a>, <a href="https://theconversation.com/institutions/university-of-technology-sydney-936">University of Technology Sydney</a> and <a href="https://theconversation.com/profiles/mark-hutchinson-105409">Mark Hutchinson</a>, <a href="https://theconversation.com/institutions/university-of-adelaide-1119">University of Adelaide</a> Have you ever wondered why your back aches when you’re down with the flu or a cold? Or COVID? This discomfort, common during many illnesses, is not just a random symptom. It’s a result of complex interactions between your immune system and your brain called the “<a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3314453/">neuroimmune synapse</a>”. A fascinating and yet-to-be-understood consequence of this conversation between the immune and brain systems during sickness is that it is particularly noticeable in the <a href="https://academic.oup.com/brain/article/145/3/1098/6370954">lower back</a>. This is thought to be one of the body’s most sensitive regions to neuroimmune threats. <h2>Immunology basics</h2> Our immune system is a double-edged sword. Yes, it fights off infections for us – but it also makes us acutely aware of the job it is doing. When our body detects an infection, our immune system releases molecules including signalling proteins called <a href="https://www.researchgate.net/publication/227831648_The_functions_of_cytokines_and_their_uses_in_toxicology">cytokines</a>. These proteins coordinate our immune system to fight off the infection and talk to our brain and spinal cord to <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2740752/#:%7E:text=Production%20of%20proinflammatory%20cytokines%20induces,to%20depression%20in%20vulnerable%20individuals.">change our behaviour</a> and physiology. This can result in symptoms like fatigue, loss of appetite, fever and increased sensitivity to pain. Classically, we think of this as a beneficial behavioural change to help us conserve energy to fight off the infection. It’s why we often feel the need to rest and withdraw from our usual activities when we’re sick – and also why we are grumpier than usual. <h2>Invisibly small changes</h2> Part of this self-protective response is a change in how we perceive threats, including sensory stimuli. When we are sick, touch can become painful and muscles can ache. Many changes in behaviour and sensory systems are <a href="https://doi.org/10.1159/000521476">believed</a> to have origins at the nanoscale. When molecular changes occur in part of the brain linked to cognition or mood, we think and feel differently. If these neuroimmune synapse changes happen in the sensory processing regions of the brain and spinal cord, we feel more pain. Such sensory changes, known as <a href="https://www.iasp-pain.org/resources/fact-sheets/allodynia-and-hyperalgesia-in-neuropathic-pain/#:%7E:text=Allodynia%20is%20pain%20due%20to,stimulus%20that%20normally%20provokes%20pain.">allodynia and hyperalgesia</a>, can lead to heightened pain sensitivity, even in areas not directly affected by the infection – <a href="https://www.sciencedirect.com/science/article/abs/pii/S0889159114001731?via%3Dihub">such as the lower back</a>. <h2>Immune memories</h2> This immune response happens with a range of bacterial infections and viruses like COVID or the flu. In fact, the sick feeling we sometimes get after a vaccination is the good work our immune system is doing to contribute to <a href="https://theconversation.com/you-cant-get-influenza-from-a-flu-shot-heres-how-it-works-118916">a protective immune memory</a>. Some of that immune-cellular conversation also alerts our brains that we are sick, or makes us think we are. After some viral infections, the sick feeling persists longer than the virus. We are seeing a long-term response to COVID in some people, termed <a href="https://theconversation.com/when-does-covid-become-long-covid-and-whats-happening-in-the-body-when-symptoms-persist-heres-what-weve-learnt-so-far-188976">long COVID</a>. Women, who generally have a <a href="https://www.nature.com/articles/nri.2016.90">stronger immune response</a> than men, may be more likely to <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7937378/">experience pain symptoms</a>. Their heightened immune response (while beneficial in resisting infections) also predisposes women to a higher risk of inflammatory conditions like <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7980266/">autoimmune diseases</a>. <h2>When to worry and what to do</h2> If the pain is severe, persistent, or accompanied by other concerning symptoms, seek medical attention. Mild to moderate pain is a common symptom during illness and we often notice this in the lower back. The good news is it usually subsides as the infection clears and the sickness resolves. While treating the underlying infection is crucial, there are also ways to dial down sickness-induced neuroimmune pain. Maintaining a diverse microbiome (the collection of microorganisms living in and on your body) by <a href="https://pubmed.ncbi.nlm.nih.gov/31704402/">eating well and getting outside</a> can help. Getting quality sleep, staying hydrated and minimising inflammation <a href="https://karger.com/bbe/article/97/3-4/197/821576/Sickness-and-the-Social-Brain-How-the-Immune">helps too</a>. Amazingly, there is <a href="https://pubmed.ncbi.nlm.nih.gov/34404209/">research</a> suggesting your grandmother’s traditional chicken broth recipe decreases the immune signals at the neuroimmune synapse. Scientists are also <a href="https://pubmed.ncbi.nlm.nih.gov/24799686/">showing</a> mindfulness meditation, cold water therapy and controlled breathing can drive profound cellular and molecular changes to help activate bodily systems like the autonomic nervous system and alter the immune response. These practices might not only help manage pain but also add an anti-inflammatory component to the immune response, reducing the severity and duration of sickness. Heat treatment (with a pack or hot water bottle) might <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8401625/">provide some relief</a> due to increased circulation. Over-the-counter pain relief maybe also be helpful but seek advice if you are taking other medications. <h2>All in the mind?</h2> Is this all mind over matter? A little of yes and a lot of no. The little of yes comes from <a href="https://pubmed.ncbi.nlm.nih.gov/26194270/">research</a> supporting the idea that if you expect your breathing, meditation and cold bath therapy to work, it may well make a difference at the cellular and molecular level. But by understanding the mechanisms of back pain during illness and by using some simple strategies, there is hope to manage this pain effectively. Always remember to seek medical help if your symptoms are severe or persist longer than expected. Your health and comfort are paramount.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/207222/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/joshua-pate-1399299">Joshua Pate</a>, Senior Lecturer in Physiotherapy, <a href="https://theconversation.com/institutions/university-of-technology-sydney-936">University of Technology Sydney</a> and <a href="https://theconversation.com/profiles/mark-hutchinson-105409">Mark Hutchinson</a>, Professor, <a href="https://theconversation.com/institutions/university-of-adelaide-1119">University of Adelaide</a> Image credits: Getty Images This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/why-does-my-back-get-so-sore-when-im-sick-the-connection-between-immunity-and-pain-207222">original article</a>.

Caring

Did COVID harm my immune system?

So you’ve had COVID and have now recovered. You don’t have ongoing symptoms and luckily, you don’t seem to have developed <a href="https://theconversation.com/long-covid-how-researchers-are-zeroing-in-on-the-self-targeted-immune-attacks-that-may-lurk-behind-it-169911" target="_blank" rel="noopener">long COVID</a>. But what impacts has COVID had on your overall immune system? It’s early days yet. But growing evidence suggests there are changes to your immune system that may put you at risk of other infectious diseases. Here’s what we know so far. <h2>A round of viral infections</h2> Over this past winter, many of us have had what seemed like a <a href="https://theconversation.com/why-do-i-and-my-kids-get-so-many-colds-and-with-all-this-covid-around-should-we-be-isolating-too-179302" target="_blank" rel="noopener">continual round</a> of viral illness. This may have included COVID, <a href="https://www.who.int/health-topics/influenza-seasonal#tab=tab_1" target="_blank" rel="noopener">influenza</a> or infection with <a href="https://www.mayoclinic.org/diseases-conditions/respiratory-syncytial-virus/symptoms-causes/syc-20353098" target="_blank" rel="noopener">respiratory syncytial virus</a>. We may have recovered from one infection, only to get another. Then there is the re-emergence of infectious diseases globally such as <a href="https://theconversation.com/we-need-to-talk-about-monkeypox-without-shame-and-blame-188295" target="_blank" rel="noopener">monkeypox</a> or <a href="https://theconversation.com/the-latest-polio-cases-have-put-the-world-on-alert-heres-what-this-means-for-australia-and-people-travelling-overseas-188989" target="_blank" rel="noopener">polio</a>. Could these all be connected? Does COVID somehow weaken the immune system to make us more prone to other infectious diseases? There are <a href="https://www.sciencedirect.com/science/article/pii/B9780128009475000168?via%3Dihub" target="_blank" rel="noopener">many reasons</a> for infectious diseases to emerge in new locations, after many decades, or in new populations. So we cannot jump to the conclusion COVID infections have given rise to these and other viral infections. But evidence is building of the negative impact of COVID on a healthy individual’s immune system, several weeks after symptoms have subsided. <h2>What happens when you catch a virus?</h2> There are three possible outcomes after a viral infection: 1) your immune system clears the infection and you recover (for instance, with <a href="https://www.healthychildren.org/English/health-issues/conditions/ear-nose-throat/Pages/Rhinovirus-Infections.aspx" target="_blank" rel="noopener">rhinovirus</a> which causes the common cold) 2) your immune system fights the virus into “latency” and you recover with a virus dormant in our bodies (for instance, <a href="https://www.healthdirect.gov.au/chickenpox" target="_blank" rel="noopener">varicella zoster virus</a>, which causes chickenpox) 3) your immune system fights, and despite best efforts the virus remains “chronic”, replicating at very low levels (this can occur for <a href="https://www.who.int/news-room/fact-sheets/detail/hepatitis-c" target="_blank" rel="noopener">hepatitis C virus</a>). Ideally we all want option 1, to clear the virus. In fact, most of us <a href="https://biosignaling.biomedcentral.com/articles/10.1186/s12964-022-00856-w" target="_blank" rel="noopener">clear SARS-CoV-2</a>, the virus that causes COVID. That’s through a complex process, using many different parts of our immune system. But international evidence suggests changes to our immune cells after SARS-CoV-2 infection may have other impacts. It may affect our ability to fight other viruses, as well as other pathogens, such as bacteria or fungi. <h2>How much do we know?</h2> An <a href="https://bmcmedicine.biomedcentral.com/articles/10.1186/s12916-021-02228-6" target="_blank" rel="noopener">Australian study</a> has found SARS-CoV-2 alters the balance of immune cells up to 24 weeks after clearing the infection. There were changes to the relative numbers and types of immune cells between people who had recovered from COVID compared with healthy people who had not been infected. This included changes to cells of the <a href="https://www.khanacademy.org/test-prep/mcat/organ-systems/the-immune-system/a/innate-immunity" target="_blank" rel="noopener">innate immune system</a> (which provides a non-specific immune response) and the <a href="https://www.ncbi.nlm.nih.gov/books/NBK21070/" target="_blank" rel="noopener">adaptive immune system</a> (a specific immune response, targeting a recognised foreign invader). <a href="https://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1009742" target="_blank" rel="noopener">Another study</a> focused specifically on <a href="https://www.immunology.org/public-information/bitesized-immunology/cells/dendritic-cells" target="_blank" rel="noopener">dendritic cells</a> – the immune cells that are often considered the body’s “first line of defence”. Researchers found fewer of these cells circulating after people recovered from COVID. The ones that remained were less able to activate white blood cells known as <a href="https://www.britannica.com/science/T-cell" target="_blank" rel="noopener">T-cells</a>, a critical step in activating anti-viral immunity. Other studies have found different impacts on T-cells, and other types of white blood cells known as <a href="https://askabiologist.asu.edu/b-cell" target="_blank" rel="noopener">B-cells</a> (cells involved in producing antibodies). After SARS-CoV-2 infection, one study <a href="https://doi.org/10.1172/JCI140491" target="_blank" rel="noopener">found evidence</a> many of these cells had been activated and “exhausted”. This suggests the cells are dysfunctional, and might not be able to adequately fight a subsequent infection. In other words, sustained activation of these immune cells after a SARS-CoV-2 infection may have an impact on other inflammatory diseases. <a href="https://www.nature.com/articles/s41392-021-00749-3#citeas" target="_blank" rel="noopener">One study</a> found people who had recovered from COVID have changes in different types of B-cells. This included changes in the cells’ metabolism, which may impact how these cells function. Given B-cells are critical for producing antibodies, we’re not quite sure of the precise implications. Could this influence how our bodies produce antibodies against SARS-CoV-2 should we encounter it again? Or could this impact our ability to produce antibodies against pathogens more broadly – against other viruses, bacteria or fungi? The study did not say. <h2>What impact will these changes have?</h2> One of the main concerns is whether such changes may impact how the immune system responds to other infections, or whether these changes might worsen or cause other chronic conditions. So more work needs to be done to understand the long-term impact of SARS-CoV-2 infection on a person’s immune system. For instance, we still don’t know how long these changes to the immune system last, and if the immune system recovers. We also don’t know if SARS-CoV-2 triggers other chronic illnesses, such as <a href="https://www.healthdirect.gov.au/chronic-fatigue-syndrome-cfs-me" target="_blank" rel="noopener">chronic fatigue syndrome</a> (myalgic encephalomyelitis). Research into this is ongoing. What we do know is that having a healthy immune system and being vaccinated (when a vaccine has been developed) is critically important to have the best chance of fighting any infection. This article originally appeared on <a href="https://theconversation.com/ive-had-covid-and-am-constantly-getting-colds-did-covid-harm-my-immune-system-am-i-now-at-risk-of-other-infectious-diseases-188899" target="_blank" rel="noopener">The Conversation</a>. Image: Shutterstock

Body

AI system sees beyond the frame of famous artworks

A new AI tool can provide a glimpse of what could potentially be going on beyond the frame of famous paintings, giving them a brand new life. OpenAI, a San Francisco-based company, has created a new tool called 'Outpainting' for its text-to-image AI system, DALL-E. Outpainting allows the system to imagine what's outside the frame of famous works such as Girl with The Pearl Earring, Mona Lisa and Dogs Playing Poker. DALL-E relies on artificial neural networks (ANNs), which simulate the way the brain works in order to learn and create an image from text. Now with Outpainting, users must describe the extended visuals in text form for DALL-E to “paint” the newly imagined artwork. Outpainting, which is primarily aimed for professionals who work with images, will let users 'extend their creativity' and 'tell a bigger story', according to OpenAI. US artist August Kamp used Outpainting to reimagine the famous 1665 painting Girl with a Pearl Earring by Johannes Vermeer, extending the background in the original style. The results show the iconic subject in a domestic setting, surrounded by crockery, houseplants, fruit, boxes and more. Other Outpainting attempts took a more creative turn, with one showing the Mona Lisa surrounded by a dystopian wasteland, and a version of A Friend In Need showing an additional table of gambling canines. <blockquote class="twitter-tweet"> “Outpainting: an apocalyptic Mona Lisa” by tonidl1989<a href="https://twitter.com/hashtag/dalle?src=hash&ref_src=twsrc%5Etfw">#dalle</a> <a href="https://twitter.com/hashtag/dalle2?src=hash&ref_src=twsrc%5Etfw">#dalle2</a> <a href="https://twitter.com/hashtag/aiart?src=hash&ref_src=twsrc%5Etfw">#aiart</a> <a href="https://twitter.com/hashtag/aiartwork?src=hash&ref_src=twsrc%5Etfw">#aiartwork</a> <a href="https://t.co/puYVxjyFMm">pic.twitter.com/puYVxjyFMm</a> — Best Dalle2 AI Art 🎨 (@Dalle2AI) <a href="https://twitter.com/Dalle2AI/status/1565168579376566278?ref_src=twsrc%5Etfw">September 1, 2022</a></blockquote> <blockquote class="twitter-tweet"> Used DALL-E 2’s new “outpainting” feature to expand the world’s greatest work of art… <a href="https://t.co/0HXQzngt9P">pic.twitter.com/0HXQzngt9P</a> — M.G. Siegler (@mgsiegler) <a href="https://twitter.com/mgsiegler/status/1565398150482784256?ref_src=twsrc%5Etfw">September 1, 2022</a></blockquote> DALL-E is available to more than one million people to create AI-generated images, all with a series of text prompts. DALL-E is just one of many AI systems infiltrating the art world, joining the likes of Midjourney and Imagen redefining how we create and appreciate art. Image credits: DALL-E - August Kamp

Art

Anne Heche's blood test results revealed after horror smash

An investigation of Anne Heche’s blood results have detected “the presence of drugs” following <a href="https://www.oversixty.com.au/news/news/new-details-of-actor-anne-heche-s-fiery-crash" target="_blank" rel="noopener">her horror smash</a>. The US actress was pulled out of her burning car after she smashed into a mansion in Los Angeles while driving a terrifying 140km/h on August 5. The 53-year-old suffered severe injuries and soon after the crash fell into a coma and has not regained consciousness since. Almost a week after the incident, a Los Angeles Police Department Public Information Officer confirmed there was a “presence of drugs” in Anne’s blood system. “Based on the blood draw, it revealed the presence of drugs, however additional testing is required to rule out any substances that were administered at the hospital as part of her medical treatment,” they told <a href="https://www.foxnews.com/entertainment/anne-heche-blood-test-revealed-presence-of-drugs" target="_blank" rel="noopener">Fox News Digital</a>. "Any secondary drugs [takes] up to 30 days for [a] secondary test to come back.” TMZ previously reported that Anne had cocaine in her system upon arrival at the hospital, but that is yet to be confirmed. The crash is currently being investigated by the LAPD who have confirmed that if Anne is found to have been drunk she would face significant charges. "If found intoxicated, [Heche] could be charged with misdemeanour DUI hit and run. No arrests have been made so far,” a representative said. Anne’s representative has confirmed that she is in an “extreme critical condition”. “She has a significant pulmonary injury requiring mechanical ventilation and burns that require surgical intervention,” her representative said. “She is in a coma and has not regained consciousness since shortly after the accident.” Image: Getty

Caring

Pay ‘with a smile or a wave’: Why Mastercard’s new face recognition payment system raises concerns

Mastercard’s <a href="https://www.mastercard.com/news/press/2022/may/with-a-smile-or-a-wave-paying-in-store-just-got-personal/" target="_blank" rel="noopener">“smile to pay”</a> system, announced last week, is supposed to save time for customers at checkouts. It is being trialled in Brazil, with future pilots planned for the Middle East and Asia. The company argues touch-less technology will help speed up transaction times, shorten lines in shops, heighten security and improve hygiene in businesses. But it raises concerns relating to customer privacy, data storage, crime risk and bias. How will it work? Mastercard’s biometric checkout system will provide customers facial recognition-based payments, by linking the biometric authentication systems of a number of third-party companies with Mastercard’s own payment systems. A Mastercard spokesperson told The Conversation it had already partnered with NEC, Payface, Aurus, Fujitsu Limited, PopID and PayByFace, with more providers to be named. <figure class="align-center zoomable"><a href="https://images.theconversation.com/files/464953/original/file-20220524-22-ga0v7l.jpeg?ixlib=rb-1.1.0&q=45&auto=format&w=1000&fit=clip"><img src="https://images.theconversation.com/files/464953/original/file-20220524-22-ga0v7l.jpeg?ixlib=rb-1.1.0&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/464953/original/file-20220524-22-ga0v7l.jpeg?ixlib=rb-1.1.0&q=45&auto=format&w=600&h=400&fit=crop&dpr=1 600w, https://images.theconversation.com/files/464953/original/file-20220524-22-ga0v7l.jpeg?ixlib=rb-1.1.0&q=30&auto=format&w=600&h=400&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/464953/original/file-20220524-22-ga0v7l.jpeg?ixlib=rb-1.1.0&q=15&auto=format&w=600&h=400&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/464953/original/file-20220524-22-ga0v7l.jpeg?ixlib=rb-1.1.0&q=45&auto=format&w=754&h=503&fit=crop&dpr=1 754w, https://images.theconversation.com/files/464953/original/file-20220524-22-ga0v7l.jpeg?ixlib=rb-1.1.0&q=30&auto=format&w=754&h=503&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/464953/original/file-20220524-22-ga0v7l.jpeg?ixlib=rb-1.1.0&q=15&auto=format&w=754&h=503&fit=crop&dpr=3 2262w" alt="The 'Fujitsu' logo in red is displayed on a building's side" /></a><figcaption>Mastercard has partnered with Fujitsu, a massive information and communications technology firm offering many different products and services. Shutterstock</figcaption></figure> They said “providers need to go through independent laboratory certification against the program criteria to be considered” – but details of these criteria aren’t yet publicly available. According to <a href="https://www.siliconrepublic.com/business/mastercard-facial-recognition-biometric-payments" target="_blank" rel="noopener">media</a> reports, customers will have to install an app which will take their picture and payment information. This information will be saved and stored on the third-party provider’s servers. At the checkout, the customer’s face will be matched with the stored data. And once their identity is verified, funds will be deducted automatically. The “wave” option is a bit of a trick: as the customer watches the camera while waving, the camera still scans their face – not their hand. Similar authentication technologies are used on smartphones (face ID) and in many airports around the world, including “<a href="https://www.abf.gov.au/entering-and-leaving-australia/smartgates/arrivals" target="_blank" rel="noopener">smartgates</a>” in Australia. <a href="https://www.theverge.com/2017/9/4/16251304/kfc-china-alipay-ant-financial-smile-to-pay" target="_blank" rel="noopener">China</a> started using biometrics-based checkout technology back in 2017. But Mastercard is among the first to launch such a system in Western markets – competing with the “pay with your palm” <a href="https://techcrunch.com/2020/09/29/amazon-introduces-the-amazon-one-a-way-to-pay-with-your-palm-when-entering-stores/" target="_blank" rel="noopener">system</a> used at cashier-less Amazon Go and Whole Foods brick and mortars in the United States. What we don’t know Much about the precise functioning of Mastercard’s system isn’t clear. How accurate will the facial recognition be? Who will have access to the databases of biometric data? A Mastercard spokesperson told The Conversation customers’ data would be stored with the relevant biometric service provider in encrypted form, and removed when the customer “indicates they want to end their enrolment”. But how will the removal of data be enforced if Mastercard itself can’t access it? Obviously, privacy protection is a major concern, especially when there are many potential third-party providers involved. On the bright side, Mastercard’s <a href="https://www.investopedia.com/articles/markets/032615/how-mastercard-makes-its-money-ma.asp" target="_blank" rel="noopener">customers</a> will have a choice as to whether or not they use the biometrics checkout system. However, it will be at retailers’ discretion whether they offer it, or whether they offer it exclusively as the only payment option. Similar face-recognition technologies used in airports, and <a href="https://www.brookings.edu/research/police-surveillance-and-facial-recognition-why-data-privacy-is-an-imperative-for-communities-of-color/" target="_blank" rel="noopener">by police</a>, often offer no choice. We can assume Mastercard and the biometrics provider with whom they partner will require customer consent, as per most privacy laws. But will customers know what they are consenting to? Ultimately, the biometric service providers Mastercard teams up with will decide how they use the data, for how long, where they store it, and who can access it. Mastercard will merely decide what providers are “good enough” to be accepted as partners, and the minimum standards they must adhere to. Customers who want the convenience of this checkout service will have to consent to all the related data and privacy terms. And as reports have noted, there is potential for Mastercard to integrate the feature with loyalty schemes and make personalised recommendations <a href="https://www.cnbc.com/2022/05/17/mastercard-launches-tech-that-lets-you-pay-with-your-face-or-hand.html" target="_blank" rel="noopener">based on purchases</a>. Accuracy is a problem While the accuracy of face recognition technologies has previously been challenged, the current best facial authentication algorithms have an error of just 0.08%, according to tests by the <a href="https://github.com/usnistgov/frvt/blob/nist-pages/reports/1N/frvt_1N_report_2020_03_27.pdf" target="_blank" rel="noopener">National Institute of Standards and Technology</a>. In some countries, even banks have <a href="https://techhq.com/2020/09/biometrics-the-most-secure-solution-for-banking/" target="_blank" rel="noopener">become comfortable</a> relying on it to log users into their accounts. Yet we can’t know how accurate the technologies used in Mastercard’s biometric checkout system will be. The algorithms underpinning a technology can work almost perfectly when trailed in a lab, but perform <a href="https://www.csis.org/blogs/technology-policy-blog/how-accurate-are-facial-recognition-systems-%E2%80%93-and-why-does-it-matter" target="_blank" rel="noopener">poorly</a> in real life settings, where lighting, angles and other parameters are varied. Bias is another problem In a 2019 study, NIST <a href="https://nvlpubs.nist.gov/nistpubs/ir/2019/NIST.IR.8280.pdf#page=5" target="_blank" rel="noopener">found</a> that out of 189 facial recognition algorithms, the majority were biased. Specifically, they were less accurate on people from racial and ethnic minorities. Even if the technology has improved in the past few years, it’s not foolproof. And we don’t know the extent to which Mastercard’s system has overcome this challenge. If the software fails to recognise a customer at the check out, they might end up disappointed, or even become irate – which would completely undo any promise of speed or convenience. But if the technology misidentifies a person (for instance, John is recognised as Peter – or <a href="https://www.youtube.com/watch?v=e8-yupM-6Oc" target="_blank" rel="noopener">twins are confused</a> for each other), then money could be taken from the wrong person’s account. How would such a situation be dealt with? <figure class="align-center zoomable"><a href="https://images.theconversation.com/files/464424/original/file-20220520-19-5hfuvx.jpeg?ixlib=rb-1.1.0&q=45&auto=format&w=1000&fit=clip"><img src="https://images.theconversation.com/files/464424/original/file-20220520-19-5hfuvx.jpeg?ixlib=rb-1.1.0&q=45&auto=format&w=754&fit=clip" sizes="(min-width: 1466px) 754px, (max-width: 599px) 100vw, (min-width: 600px) 600px, 237px" srcset="https://images.theconversation.com/files/464424/original/file-20220520-19-5hfuvx.jpeg?ixlib=rb-1.1.0&q=45&auto=format&w=600&h=617&fit=crop&dpr=1 600w, https://images.theconversation.com/files/464424/original/file-20220520-19-5hfuvx.jpeg?ixlib=rb-1.1.0&q=30&auto=format&w=600&h=617&fit=crop&dpr=2 1200w, https://images.theconversation.com/files/464424/original/file-20220520-19-5hfuvx.jpeg?ixlib=rb-1.1.0&q=15&auto=format&w=600&h=617&fit=crop&dpr=3 1800w, https://images.theconversation.com/files/464424/original/file-20220520-19-5hfuvx.jpeg?ixlib=rb-1.1.0&q=45&auto=format&w=754&h=776&fit=crop&dpr=1 754w, https://images.theconversation.com/files/464424/original/file-20220520-19-5hfuvx.jpeg?ixlib=rb-1.1.0&q=30&auto=format&w=754&h=776&fit=crop&dpr=2 1508w, https://images.theconversation.com/files/464424/original/file-20220520-19-5hfuvx.jpeg?ixlib=rb-1.1.0&q=15&auto=format&w=754&h=776&fit=crop&dpr=3 2262w" alt="" /></a><figcaption>There’s no evidence facial recognition technology is infallible. These systems can misidentify and also have biases. Shutterstock</figcaption></figure> Is the technology secure? We often hear about software and databases being hacked, even in <a href="https://www.csoonline.com/article/2130877/the-biggest-data-breaches-of-the-21st-century.html" target="_blank" rel="noopener">cases of</a> supposedly very “secure” organisations. Despite Mastercard’s <a href="https://wwmastw.cnbc.com/2022/05/17/mastercard-launches-tech-that-lets-you-pay-with-your-face-or-hand.html" target="_blank" rel="noopener">efforts</a> to ensure security, there’s no guarantee the third-party providers’ databases – with potentially millions of people’s biometric data – won’t be hacked. In the wrong hands, this data could lead to <a href="https://www.comparitech.com/identity-theft-protection/identity-theft-statistics/" target="_blank" rel="noopener">identity theft</a>, which is one of the fastest growing types of crime, and financial fraud. Do we want it? Mastercard suggests 74% of customers are in favour of using such technology, referencing a stat from its <a href="https://www.mastercard.com/news/ap/en/newsroom/press-releases/en/2020/april/mastercard-study-shows-consumers-moving-to-contactless-payments-for-everyday-purchases/" target="_blank" rel="noopener">own study</a> – also used by <a href="https://www.mastercard.com/news/ap/en/newsroom/press-releases/en/2020/october/mastercard-idemia-and-matchmove-pilot-fingerprint-biometric-card-in-asia-to-enhance-security-and-safety-of-contactless-payments" target="_blank" rel="noopener">business partner</a> Idemia (a company that sells biometric identification products). But the report cited is vague and brief. Other studies show entirely different results. For example, <a href="https://www.getapp.com/resources/facial-recognition-technology/#how-comfortable-are-consumers-with-facial-recognition-technology" target="_blank" rel="noopener">this study</a> suggests 69% of customers aren’t comfortable with face recognition tech being used in retail settings. And <a href="https://www.securitymagazine.com/articles/93521-are-consumers-comfortable-with-facial-recognition-it-depends-says-new-study" target="_blank" rel="noopener">this one</a> shows only 16% trust such tech. Also, if consumers knew the risks the technology poses, the number of those willing to use it might drop even lower.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/183447/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/rita-matulionyte-170113" target="_blank" rel="noopener">Rita Matulionyte</a>, Senior Lecturer in Law, <a href="https://theconversation.com/institutions/macquarie-university-1174" target="_blank" rel="noopener">Macquarie University</a> This article is republished from <a href="https://theconversation.com" target="_blank" rel="noopener">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/pay-with-a-smile-or-a-wave-why-mastercards-new-face-recognition-payment-system-raises-concerns-183447" target="_blank" rel="noopener">original article</a>. Image: Getty Images

Technology

”Fix the system not me”: A first for Australian Fashion Week

In a first for the yearly week-long display of new Aussie fashion, adaptive clothing has taken the fore through a show solely focused on the underrepresented sphere of fashion. Day four of <a href="https://aafw.com.au/index" target="_blank" rel="noopener">Australian Fashion Week</a> saw a collective of designers take to the runway with fashionable garments that can be worn by both abled and disabled people and were modelled by the very people who inspired them. JAM and Christine Stephens, two designers whose designs were featured, share a mutual goal: to act as a voice for the adaptive designers collective and highlight the possibilities for stylish fashion that is inclusive. <blockquote class="instagram-media" style="background: #FFF; border: 0; border-radius: 3px; box-shadow: 0 0 1px 0 rgba(0,0,0,0.5),0 1px 10px 0 rgba(0,0,0,0.15); margin: 1px; max-width: 540px; min-width: 326px; padding: 0; width: calc(100% - 2px);" data-instgrm-captioned="" data-instgrm-permalink="https://www.instagram.com/p/CdcVj66rDK8/?utm_source=ig_embed&utm_campaign=loading" data-instgrm-version="14"> <div style="padding: 16px;"> <div style="display: flex; flex-direction: row; align-items: center;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 40px; margin-right: 14px; width: 40px;"> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 100px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 60px;"> </div> </div> </div> <div style="padding: 19% 0;"> </div> <div style="display: block; height: 50px; margin: 0 auto 12px; width: 50px;"> </div> <div style="padding-top: 8px;"> <div style="color: #3897f0; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: 550; line-height: 18px;">View this post on Instagram</div> </div> <div style="padding: 12.5% 0;"> </div> <div style="display: flex; flex-direction: row; margin-bottom: 14px; align-items: center;"> <div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(0px) translateY(7px);"> </div> <div style="background-color: #f4f4f4; height: 12.5px; transform: rotate(-45deg) translateX(3px) translateY(1px); width: 12.5px; flex-grow: 0; margin-right: 14px; margin-left: 2px;"> </div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(9px) translateY(-18px);"> </div> </div> <div style="margin-left: 8px;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 20px; width: 20px;"> </div> <div style="width: 0; height: 0; border-top: 2px solid transparent; border-left: 6px solid #f4f4f4; border-bottom: 2px solid transparent; transform: translateX(16px) translateY(-4px) rotate(30deg);"> </div> </div> <div style="margin-left: auto;"> <div style="width: 0px; border-top: 8px solid #F4F4F4; border-right: 8px solid transparent; transform: translateY(16px);"> </div> <div style="background-color: #f4f4f4; flex-grow: 0; height: 12px; width: 16px; transform: translateY(-4px);"> </div> <div style="width: 0; height: 0; border-top: 8px solid #F4F4F4; border-left: 8px solid transparent; transform: translateY(-4px) translateX(8px);"> </div> </div> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center; margin-bottom: 24px;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 224px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 144px;"> </div> </div> <p style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; line-height: 17px; margin-bottom: 0; margin-top: 8px; overflow: hidden; padding: 8px 0 7px; text-align: center; text-overflow: ellipsis; white-space: nowrap;"><a style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: normal; line-height: 17px; text-decoration: none;" href="https://www.instagram.com/p/CdcVj66rDK8/?utm_source=ig_embed&utm_campaign=loading" target="_blank" rel="noopener">A post shared by Australian Fashion Week (@ausfashionweek)</a> </div> </blockquote> “We want to give people living with disabilities a choice. A choice in fashion, a choice to be included and a choice to be heard,” Jessie Sadler and Carol Taylor, two designers for Christine Stephens, said in a press release. Taylor, the co-owner and lead designer of the fashion label, said the collection was inspired by a quadriplegic groom-to-be who “would never know what it would be like to unwrap his able-bodied bride”. <img src="https://oversixtydev.blob.core.windows.net/media/2022/05/fashion-week1.jpg" alt="" width="1280" height="720" /> Models with disabilities took to the runway for the Adaptive Clothing Collective show. Image: Getty Images The outfits included multiple adaptive features, such as particularly magnetic button closures and zips to improve access for prosthetic users and even particularly contrasting colour choices. One model’s outfit, a sky-blue two-piece suit with contrasting magenta collars and inner linings, was inspired by their visual impairment that prevents them from distinguishing between darker colours, as reported by <a href="https://www.elle.com.au/fashion/adaptive-clothing-collective-australian-fashion-week-2022-27051" target="_blank" rel="noopener">Elle Australia</a>. The runway featured disabled models throughout the show, including actress Chloé Hayden, who told <a href="https://www.refinery29.com/en-au/2022/05/10974587/adaptive-fashion-runway" target="_blank" rel="noopener">Refinery29</a> being asked to participate was an “out of this world” experience. “I grew up practising my model walk, doing personal little photo shoots, going to those Dolly model searches … I’m always jaw dropping at Australian Fashion Week,” she told the publication. <blockquote class="instagram-media" style="background: #FFF; border: 0; border-radius: 3px; box-shadow: 0 0 1px 0 rgba(0,0,0,0.5),0 1px 10px 0 rgba(0,0,0,0.15); margin: 1px; max-width: 540px; min-width: 326px; padding: 0; width: calc(100% - 2px);" data-instgrm-captioned="" data-instgrm-permalink="https://www.instagram.com/p/CdcszIUBLKa/?utm_source=ig_embed&utm_campaign=loading" data-instgrm-version="14"> <div style="padding: 16px;"> <div style="display: flex; flex-direction: row; align-items: center;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 40px; margin-right: 14px; width: 40px;"> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 100px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 60px;"> </div> </div> </div> <div style="padding: 19% 0;"> </div> <div style="display: block; height: 50px; margin: 0 auto 12px; width: 50px;"> </div> <div style="padding-top: 8px;"> <div style="color: #3897f0; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: 550; line-height: 18px;">View this post on Instagram</div> </div> <div style="padding: 12.5% 0;"> </div> <div style="display: flex; flex-direction: row; margin-bottom: 14px; align-items: center;"> <div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(0px) translateY(7px);"> </div> <div style="background-color: #f4f4f4; height: 12.5px; transform: rotate(-45deg) translateX(3px) translateY(1px); width: 12.5px; flex-grow: 0; margin-right: 14px; margin-left: 2px;"> </div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(9px) translateY(-18px);"> </div> </div> <div style="margin-left: 8px;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 20px; width: 20px;"> </div> <div style="width: 0; height: 0; border-top: 2px solid transparent; border-left: 6px solid #f4f4f4; border-bottom: 2px solid transparent; transform: translateX(16px) translateY(-4px) rotate(30deg);"> </div> </div> <div style="margin-left: auto;"> <div style="width: 0px; border-top: 8px solid #F4F4F4; border-right: 8px solid transparent; transform: translateY(16px);"> </div> <div style="background-color: #f4f4f4; flex-grow: 0; height: 12px; width: 16px; transform: translateY(-4px);"> </div> <div style="width: 0; height: 0; border-top: 8px solid #F4F4F4; border-left: 8px solid transparent; transform: translateY(-4px) translateX(8px);"> </div> </div> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center; margin-bottom: 24px;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 224px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 144px;"> </div> </div> <p style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; line-height: 17px; margin-bottom: 0; margin-top: 8px; overflow: hidden; padding: 8px 0 7px; text-align: center; text-overflow: ellipsis; white-space: nowrap;"><a style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: normal; line-height: 17px; text-decoration: none;" href="https://www.instagram.com/p/CdcszIUBLKa/?utm_source=ig_embed&utm_campaign=loading" target="_blank" rel="noopener">A post shared by Chloé Hayden (@chloeshayden)</a> </div> </blockquote> Following her appearance in the show, where she wore olive utility-style boots and a trench coat with the slogan ‘Fix the system, not me’, Hayden took to Instagram to share clips and her excitement. “Australian Fashion Week’s first adaptive runway. Let it not be the last,” she captioned the post. “More posts to come once I stop projectile crying and stimming.” Lisa Cox, an author, consultant and disability influencer who modelled for Christina Stephens, said the opportunity made her feel “seen” by the industry. “I feel more accepted and I feel seen by an industry that I haven’t felt a part of since I became disabled over 16 years ago … I feel both honoured and privileged to be part of such a landmark event in Australian fashion history,” she told Refinery29. Cox added that the absence of inclusivity in fashion has left her feeling unimportant and uncatered for - which she noted was also bad for those in the clothing business. “[It’s] offensive and it really hurts,” she said. “But on the other hand, people with disabilities are consumers too so that attitude is just bad business. My legs don’t work but my credit card does!” Emma Clegg and Molly Rogers, the creators of JAM the label, said the “universal design” of adaptive clothing is more comfortable, convenient and accessible for everyone, and encourage non-disabled people to also give them a try. "Purchasing from and supporting Australian inclusive [and] adaptive fashion brands will prove to the Australian fashion industry that there is the demand out there, and they need to be including people with disability throughout their businesses,” they told Refinery29. “It shouldn't be newsworthy that there is a designer with disabilities or clothes for people with disabilities at AAFW, but for now it must be celebrated,” Cox said. Whether you approach the issue from a position of fairness or business, with 20 percent of Australians living with a disability, it makes sense to design clothing that can be worn and enjoyed by everyone - and hopefully this show is just the first of many more. Here's a look at the styles appearing at the show. Image: Getty Images

Beauty & Style

"Australian GeoGRAPHIC!": Hilarious echidna pic breaks the internet

A hilarious yet graphic photo of an echidna’s penis has shocked the internet after randomly popping up in their news feed. The photo was uploaded by Australian Geographic asking readers if they had ever seen an echidna’s penis before. The photo in question shows a four-tipped penis with scientists looking into why the mammal’s reproductive system is the way it is. “Have you ever seen an echidna’s penis?” the post read. “You’ve probably asked your friends this in jest, but scientists are working away at unlocking the mystery of the bizarre, four-tipped reproductive system. “Labelled one of the ‘weirdest penises of the animal kingdom’, it's bright red and has four heads. “Now there's a dinner conversation starter…” The internet’s reaction was lighthearted, with many in the comment section leaving funny remarks. <iframe style="overflow: hidden; border: initial none initial;" src="https://www.facebook.com/plugins/post.php?href=https%3A%2F%2Fwww.facebook.com%2Fausgeo%2Fposts%2F10158869200833339&show_text=true&width=500" width="500" height="780" frameborder="0" scrolling="no" allowfullscreen="allowfullscreen"></iframe> “Such prickly little buggers obviously they need all the help they can get,” someone joked. “Australian GeoGRAPHIC,” the best comment read. “I'm guessing it's for Russian roulette impregnation,” another commented. “I cannot unsee this. Scarred for life,” someone else joked. “Wears condoms. They fit like a glove,” another comment read. “If I were a female echidna, I’d shuffle away as fast as I could from this thing and if I couldn’t out shuffle it, I’d bury myself as quickly as possible,” someone wrote. Despite the funny comments, Australian Geographic linked back to a <a href="https://www.australiangeographic.com.au/topics/wildlife/2021/05/echidna-penises-why-theyre-so-weird/?fbclid=IwAR3kQEzjVHQDodmTLm7FbqgH7ixIxcm8V0UFNzIHx9xgXofvsqkJaCc2tHI" target="_blank" rel="noopener">research article</a> about why echidna penises are weird. ”We’re not really sure why it looks so weird but we do know that they only use their penis for mating, not urine,” reproductive biologist Jane Fenelon said. “Because they don’t need it for urine, they had the freedom to make it much more elaborate and this is something you see in other species that only use it for mating.” Images: Australian Geographic

Family & Pets

Basic tips for staying healthier throughout winter

The cooler months are just around the corner and the last thing we want is to not feel our ultimate best. We can all admit that we’d rather be in bed under a warm blanket, watching TV, bingeing on junk food rather than going out to the gym, or making healthy foods because takeout is so much easier. However, it is just as important to stay fit and healthy in winter as it is in summer. Below are the Top Six ways to do just that. <ol> <li dir="ltr" aria-level="1"> Eat immune boosting foods </li> </ol> To ensure an ultimate healthy lifestyle, it is recommended to stick to foods from the five food groups. Need a refresher? They include: vegetables and legumes/beans, fruit, lean meat, fish, poultry, grains and cereal, milk, cheese and yoghurt. Stay on top of your Vitamin C which is in most fruits and veggies. Zinc is just as important and could be found in most animal products, nuts and seeds. Iron is also one to stay on top of which can be found in meat, beans, lentils and even spinach. <ol start="2"> <li dir="ltr" aria-level="1"> Drink water </li> </ol> Water is just as important in the cooler months as it is in the warmer seasons. Stay hydrated however is not just limited to water, you can drink as much soup, tea as you want and it all helps. <ol start="3"> <li dir="ltr" aria-level="1"> Stay active </li> </ol> The weather may not be in your favour but there are many fun indoor activities you can get up to. Indoor rock climbing, obstacle courses, dance class…whatever tickles your fancy. Or just join the gym. <ol start="4"> <li dir="ltr" aria-level="1"> Limit the alcohol intake </li> </ol> Alcohol affects our mood, sleep and energy levels which in turn can leave us feeling more stressed and exhausted. It also increases the risk of having a stroke, high blood pressure, heart disease, liver disease and cancers. We’re not saying to give it up completely, but it will help a lot if you reduce how much you drink. <ol start="5"> <li dir="ltr" aria-level="1"> Practice good hygiene </li> </ol> After three years of the pandemic this should be second nature. <ol start="6"> <li dir="ltr" aria-level="1"> Get some Vitamin D </li> </ol> Take advantage of the good weather whenever it arrives. When you see the sun, pop on some sunscreen, shades and hat and go out for a walk in the park, at the beach or grab a coffee with some friends. Image: Shutterstock

Body

13 foods doctors eat when they have a cold

That age-old advice is dead-on: when you’re sick, your body needs extra fuel in the form of kilojoules to defend against germs and mount an immune response. “What you eat can be one of the best weapons in your arsenal,” says pulmonologist, Dr Gustavo Ferrer. The right nutrients can give your immune system a much-needed boost, help clear up some of those irritating symptoms, and get you back on your feet faster. Doctors shared their favourite home remedies when they feel a cold coming on – and some of their recommendations may surprise you. Chicken soup It’s not just for your soul – chicken soup may actually help fight upper respiratory tract infections, according to an older, but classic study published in the journal Chest that looked at how immune cells responded in the laboratory. “When we get attacked by a virus, our natural defences can cause an inflammatory response,” says Dr Ferrer. Some combination of ingredients in the soup may inhibit the white blood cell activity that can cause inflammation. Strawberries These berries are brimming with antioxidant power – they’re among the top 20 richest fruits when it comes to free radical slaying compounds – particularly vitamin C. And last year, a study in the journal Nutrients found that getting up to eight grams of vitamin C a day can help shorten the length of a cold. Adding a cup of sliced strawberries to your smoothie will net you about 100 milligrams of vitamin C. Garlic This potent bulb contains a compound known as allicin, which has antibacterial and antiviral properties that may help fight off the common cold. Chopping or crushing garlic activates this compound, so for maximum results, prep it and let it sit for several minutes before eating it. In research published in the Cochrane Database of Systemic Reviews, people who took a garlic supplement daily reported fewer colds over a three-month span than those who popped a placebo. Honey Sore throat? The sticky, sweet nectar was found to be superior to many over-the-counter cough suppressants, Dr Ferrer says. For a soothing elixir that’s the bee’s knees, combine honey with lemon juice for a little vitamin C antioxidant action, he adds. Carrots These may be one of the ingredients that give chicken soup its curative properties. Carrots and other orange-hued produce contain beta-carotene. Your body converts this compound to vitamin A, a building block of your immune system, says Dr Eudene Harry. Onions These aromatics contain quercetin, an antioxidant with antiviral and anti-inflammatory properties. “A lot of the symptoms you get when you’re sick is your body hyper-responding to things that are not necessarily a threat,” says Dr Harry. “Antioxidants help quell some of that response so it doesn’t get out of control. They remove waste products and reduce inflammation,” she says. Capsicum Research shows that being cold may suppress your immune system a bit, but vitamin C has natural immune-boosting properties. Loading up on C-rich foods can be a good way to compensate for a suppressed immune function during the chillier months, Dr Harry says. A cup of chopped capsicum has two times as much vitamin C as a medium orange. Green tea Fluids are important when you’re sick because, Dr Harry says, “Everything works better when you’re hydrated.” Green tea is a good choice because it contains the potent antioxidants catechins, along with theanine, an amino acid. Both have been linked to immunity, research shows. Ginger If the dreaded stomach flu is going around, stock up on this relieving root. It’s known for its anti-nausea properties. It also contains compounds known as gingerols that have an anti-inflammatory effect, which makes it an ideal addition to a cup of tea for soothing a sore throat. Dr Harry likes to take hers with ginger, pomegranate and lemon. Mushrooms These fungi are one of the few edible sources of vitamin D, which we normally get from the sun. Shorter daylight hours during the autumn and winter months can mean we’re not getting enough of that nutrient, which can open the door to lowered immunity. Mushrooms don’t naturally contain that much vitamin D, but if they are grown in the presence of ultraviolet light they will produce more. (Check the package for actual content of vitamin D.) Shiitake mushrooms, in particular, may help in this regard, says associate professor of medicine, Dr Gerard Mullin. A study published in the Journal of the American College of Nutrition showed that people who ate 5 or 10 grams of shiitake mushrooms a day had improved markers of immunity, including better-functioning gamma delta T cells and reductions in inflammatory proteins in their blood. Hot sauce A big hit of wasabi or sriracha can feel like it’s clearing your sinuses even when you’re not sick – and it turns out, that could very well be the case. A Cochrane review of research on nasal sprays containing capsaicin, the active compound in chilli peppers and other spicy foods, improves symptoms of nonallergic rhinitis (aka sneezing, congestion) better than a placebo, possibly because of its anti-inflammatory effect. Plus, spicy peppers are another good source of vitamin C. Turmeric Spiking your coffee with this spice, aka a golden latte, won’t hurt. Curcumin, the active compound in the vibrant yellow powder, has anti-inflammatory, antibacterial and antiviral properties. “Oxidative stress goes up when your body is under attack,” says Dr Mullin. That can cause residual damage beyond what the invading germs cause. Turmeric, he says, can help with that. YoghurtFermented dairy products may not be your go-to when you’re feeling under the weather, but they should be, says Dr Mullin. “Most of your immune system is in your gut,” he explains, and probiotic-containing foods like yoghurt help keep your gastrointestinal tract teeming with healthy bacteria. Indeed, research has found that the benefits go beyond digestive health: a 2017 study in the journal Nutrients found that eating yoghurt daily increase the activity of killer T cells, whose job it is to destroy infected cells in the body. And there’s no evidence that dairy creates phlegm or contributes to congestion, so spoon away! This article originally appeared on <a href="https://www.readersdigest.com.au/healthsmart/13-foods-doctors-eat-when-they-have-a-cold" target="_blank" rel="noopener">Reader’s Digest</a>.

Body

Grandma's hilarious ranking system goes viral

Many suspect their grandma may have favourites, but one family knows for sure where they stand thanks to their grandmother’s brutally honest "leaderboard". US comedian Dan LaMorte posted on TikTok explaining the unique feature in his grandmother's house, where at any time family members can look to see how beloved they are in their matriarch's eyes – and heart. "At my grandma’s house she has a board of the ten grandchildren's faces on magnets, and she ranks us from one to ten. I'm eight," he says proudly before cutting the video off. The comedian's TikTok followers found the system hilariously honest. "I would kill for this kind of transparency in my relationships," said one commenter. "I would have told her that she my #2 grandma," added another. "My grandma used to rank us too. She would regularly tell us of our rankings. She's been gone for 4 years, but I talk about her a lot in therapy," said one commenter. "That was the basis behind this. She used to do verbal rankings so we made her a board to do it visually," revealed LaMorte. In a separate video, the comedian displays the rankings again three months on, explaining some of the reasoning behind the shifts in order. He reveals that the number one favourite grandchild lives next door so she moves her ranking herself and is therefore invalid, while number two is a dentist and number three is a doctor. He also adds that he moved up two places, now at sixth place out of the ten grandchildren. "The key is to coast and wait for the others to mess up. The long game," one commenter suggested. "My great-grandmother's favorite grandchild is my childhood dog, whose portrait takes center stage in her home," said another. Image: TikTok

Family & Pets

I just recovered from Omicron – how long does my immunity last?

<div class="copy"> If you recently recovered from an <a rel="noreferrer noopener" href="https://cosmosmagazine.com/health/covid/omicron-update-170122/" target="_blank">Omicron infection</a>, do you now have free rein to do whatever you want without risk of reinfection? And if so, for how long? Two main factors affect how well your acquired immunity after Omicron infection will protect you. First, your antibody levels. “If high levels of neutralising antibodies are elicited to Omicron following infection, then we would expect to see some level of protection against reinfection with Omicron, but this is likely to be short-lived,” says Professor Gilda Tachedjian, a virologist at the Burnet Institute and past president of the Australian Virology Society. Generally, a more severe infection generates a higher level of antibodies than an asymptomatic infection, explains Professor Anthony Cunningham, an infectious diseases physician and clinical virologist at the University of Sydney. But when the level of neutralising antibodies begins to drop, your likelihood of reinfection rises. It’s simply too early to know how long Omicron immunity will last, he says. From vaccine studies, we know that antibody levels begin to drop after three to six months. A <a rel="noreferrer noopener" href="https://www.thelancet.com/journals/lanmic/article/PIIS2666-5247(21)00219-6/fulltext" target="_blank">recent study</a> published in The Lancet estimated that reinfection by SARS-CoV-2 under endemic conditions would likely occur between three months and five years after peak antibody response. Almost certainly, Cunningham says, there will be a lot of individual variation, similar to what has been observed with previous strains. This variation depends on the severity of the disease you experienced, and whether you have had a vaccine. The second factor: emerging variants. Our waning antibodies may not be able to target any new variants that come along. The Omicron variant, for example, largely evades immunity from past infection and vaccines. A recent <a rel="noreferrer noopener" href="https://www.imperial.ac.uk/mrc-global-infectious-disease-analysis/covid-19/report-49-Omicron/" target="_blank">report</a> from Imperial College London estimates that the risk of reinfection with Omicron is 5.4 times greater than that of the Delta variant. “The most likely outcome is that you won’t get re-infected with Omicron because the expectation is that the Omicron wave will decline, but then the greatest risk is that another strain comes along,” says Cunningham. “It all depends on what type of strain comes next.” Even if you have had a recent Omicron infection, don’t throw your mask away, warns Cunningham: “The more virus circulates in the world, the more likely it is that we’ll see new strains.” Although protection from reinfection might not last for long, experts think T-cells might come to the rescue. While antibodies directly bind and neutralise virions, preventing an infection, T-cells activate once the infection is established. They target and kill virus-infected cells, helping to clear the infection and reduce its severity. This arm of the immune response tends to be broader than antibodies – and thus more likely to recognise variants, and to last longer, Tachedjian explains. “Hopefully, you will be asymptomatic or have a less severe disease [the second time around].” Image credits: Getty Images </div> <div id="contributors"> This article was originally published on <a href="https://cosmosmagazine.com/health/covid/immunity-after-omicron-infection/">cosmosmagazine.com</a> and was written by Dr Manuela Callari. </div>

Body

Meditation could boost your immune system

A <a rel="noopener" href="https://www.scimex.org/newsfeed/how-meditation-could-help-boost-the-immune-system" target="_blank">new study</a> has connected intensive meditation with altered behaviour of over 200 genes tied to immunity, with their findings suggesting that meditation may be beneficial for those with a weakened immune system. The research, recently published in the journal <a rel="noopener" href="https://www.pnas.org/content/118/51/e2110455118" target="_blank">Proceedings of the National Academy of Sciences (PNAS)</a>, involved analysing blood samples from 106 volunteers who participated in an intensive Samyama meditation retreat. The participants spent eight days in complete silence, followed a strict vegan diet and regular sleep schedule, and meditated for more than ten hours each day. Several samples were taken from each participant, including one two months before the retreat, another five weeks before, two immediately before and after the retreat, and a final sample three months after it ended. After analysing the gene expression in the samples, the team found that there were “distinct” alterations in how genes were expressed after participants meditated. In particular, they found that 220 genes tied to immunity had higher levels of expression without an increase in inflammation. Sixty-eight of these genes were also tied to signalling proteins called interferons. These proteins help our immune systems to identify viruses and trigger immune cells to fight viral cells, stopping them from multiplying. “These findings suggest that meditation has an immediate impact on immune cells and genes,” the authors wrote. They also discovered that there wasn’t an increase in inflammation Though many studies have found that meditation has a positive impact on our health, this new research investigates what happens inside human cells when we meditate to explain why we experience these positive effects. By examining the gene expression before and after meditation, the authors suggest that meditation could be helpful for treating conditions characterised by a weakened immunity and persistent inflammation. “Together, these results make meditation an effective behavioural intervention for treating various conditions associated with a weakened immune system,” they concluded. Image: Getty Images

Mind

Can mental healthcare be automated?

Depression is predicted to become the <a rel="noreferrer noopener" href="https://journals.plos.org/plosmedicine/article?id=10.1371/journal.pmed.0030442" target="_blank">leading global cause of loss of life years</a> due to illness by 2030, yet <a rel="noreferrer noopener" href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5288082/" target="_blank">fewer than one in five people</a> who suffer depression receive appropriate care. And there are <a rel="noreferrer noopener" href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(21)02143-7/fulltext" target="_blank">worrisome signs</a> the COVID-19 pandemic is exacerbating triggers of the disease. As the burden of disease rises around the world, mental healthcare systems, <a rel="noreferrer noopener" href="https://www.who.int/news/item/08-10-2021-who-report-highlights-global-shortfall-in-investment-in-mental-health" target="_blank">many of which are already patently inadequate</a>, will be stretched thin. That’s why many experts are turning to digital interventions to help manage surging demand, packaging up psychotherapeutic treatments into computer programs and apps that can be used at home. But how effective are digital interventions? And will people accept therapy without a human face? These are the questions an international team of researchers from Finland, Germany, the Netherlands and Italy sought to answer through a systematic review and meta-analysis <a rel="noreferrer noopener" href="https://doi.org/10.1037/bul0000334" target="_blank">published today</a> in Psychological Bulletin. The team analysed 83 studies published between 1990 and 2020, reporting on 15,530 individuals, making it the largest and most comprehensive analysis of digital mental healthcare to date. The findings, while mixed, are promising. Software alone not enough The data suggests that digital interventions are effective in the treatment of depression, but the best results come when a digital program is augmented by support from an actual human. That’s when digital therapy can actually rival the effectiveness of face-to-face therapy. “Digital interventions could provide a viable, evidence-based method of meeting the growing demand for mental healthcare, especially where people are unable to access face-to-face therapy due to long waiting lists, financial constraints or other barriers,” says Isaac Moshe, lead author of the study and a PhD researcher at the University of Helsinki. But, he notes, “software alone just isn’t enough for many people, especially individuals who suffer from moderate or more severe symptoms.” Interestingly, the researchers found that while a level of human support behind a digital program was important, there was no marked difference in outcomes whether that support was provided by a highly experienced clinician or someone with less experience, such as a student or trainee. Moshe says that means digital programs could be scaled up by relying on less experienced practitioners, and offer a powerful solution to a growing problem. Even with the assistance of a clinician, however, there are barriers to the uptake of digital healthcare. According to <a rel="noreferrer noopener" href="https://www.pharmaceutical-technology.com/news/cost-security-concerns-and-lack-of-integration-into-existing-workflow-the-main-barriers-to-the-adoption-of-digital-health-tools-poll/" target="_blank">one industry-based poll</a>, major barriers include cost, security concerns and a lack of digital savviness among patients. Another major therapeutic concern is the idea that spending time working face-to-face with a human builds trust and a sense of alliance. This is <a rel="noreferrer noopener" href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7016304/" target="_blank">particularly true</a> among older generations. Digital healthcare is also generally only appropriate for those who can afford the means to access it through a mobile phone or computer. That means it’s inaccessible for many people living in poverty or in remote communities. AI has a role to play The researchers also say artificial intelligence may have a role to play, principally in flagging risk factors for mental health, as well as helping clinicians develop tailor-made interventions. “Over three billion people now own a smartphone and wearable devices are growing in popularity,” explains Lasse Sander from the University of Freiburg, who led the research team. “These devices produce a continuous stream of data related to a person’s behaviour and physiology. With new developments in artificial intelligence and machine learning, we now have promising methods of using this data to identify if someone is at risk of developing a mental illness.” Moshe cautions that the results are focused on moderate depression, and that digital interventions may not be sufficient to cater to severe cases. “There are very few studies involving people with severe depression or individuals at risk of suicide, leaving the evidence unclear for the role of digital interventions for the treatment of severe and complex depression,” he says.  <img id="cosmos-post-tracker" style="opacity: 0; height: 1px!important; width: 1px!important; border: 0!important; position: absolute!important; z-index: -1!important;" src="https://syndication.cosmosmagazine.com/?id=176089&title=Can+mental+healthcare+be+automated%3F" alt="" width="1" height="1" />  <div id="contributors"> <a rel="noopener" href="https://cosmosmagazine.com/health/body-and-mind/can-digital-mental-healthcare-be-automated/" target="_blank">This article</a> was originally published on <a rel="noopener" href="https://cosmosmagazine.com" target="_blank">Cosmos Magazine</a> and was written by <a rel="noopener" href="https://cosmosmagazine.com/contributor/amalyah-hart" target="_blank">Amalyah Hart</a>. Amalyah Hart is a science journalist based in Melbourne. Image: Getty Images </div>

Mind

Older Australians are already bamboozled by a complex home-care system. So why give them more of the same?

More than <a href="https://www.aihw.gov.au/reports/australias-welfare/aged-care">a million older Australians</a> need care at home each year. <a href="https://www.health.gov.au/sites/default/files/documents/2021/08/ninth-report-on-the-funding-and-financing-of-the-aged-care-industry-july-2021.pdf">More than 1,000 agencies</a> provide services to them. Despite the federal government allocating <a href="https://www.health.gov.au/initiatives-and-programs/aged-care-reforms/a-generational-plan-for-aged-care">significant extra funds to home care</a> in the last budget, there is still a raft of problems with current home-care arrangements. As we show in <a href="https://grattan.edu.au/report/unfinished-business-practical-policies-for-better-care-at-home/">our new report</a>, “Unfinished business: practical policies for better care at home”, the federal government is placing too much emphasis on expanding the market of services, and not enough on supporting people to access timely and quality services. Home care support ranges from help with personal care and cleaning the house, to provision of mobility aids, and transport to social events and medical appointments. People who need care at home can explore options via the federal government’s <a href="https://www.myagedcare.gov.au/">myagedcare</a> website. Then they can get assessed, find a local provider to suit their needs, and manage their own care. But this system is <a href="https://www.ingentaconnect.com/contentone/tpp/ijcc/2020/00000004/00000003/art00006">impersonal and cumbersome</a>. Assessment of people’s needs is divorced from planning their services. Older people get little advice and support to find services, and people who need more intensive and complex care often have to wait for more than a year. Administrative and coordination costs for the <a href="https://www.aihw.gov.au/reports/australias-welfare/aged-care">200,000 people who get home care packages</a> are high, hourly service charges are unregulated, and there is more than <a href="https://www.stewartbrown.com.au/images/documents/StewartBrown_-_ACFPS_Financial_Performance_Sector_Report_June_2021.pdf">A$1.6 billion in unspent funds</a> that could be used to provide services. The number of private services has grown dramatically, with little oversight of quality and value for money. At the same time, home-care workers <a href="https://www.smh.com.au/politics/federal/canberra-told-to-pay-up-to-get-aged-care-workers-a-25-per-cent-rise-20210705-p586x9.html">remain poorly paid and under-valued</a>. Training is patchy, work is often insecure, and there’s insufficient supervision, support and staff development. Not surprisingly, it is increasingly difficult to recruit and retain aged-care workers. <h2>What’s wrong with the extra funding?</h2> The federal government’s response to the landmark <a href="https://agedcare.royalcommission.gov.au/">Royal Commission into Aged Care</a> was substantial, but it doesn’t change the fundamentals of the home-care system. It expands a market that is not working for older people. The government is putting its faith in a centrally regulated market model, dominated by private and non-government home-care businesses. Even with the massively increased home-care funding, the market may still not provide enough to reduce waiting times for services to less than a month, as the royal commission recommended. <a href="https://images.theconversation.com/files/436568/original/file-20211209-137612-sxaiia.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=1000&fit=clip"><img src="https://images.theconversation.com/files/436568/original/file-20211209-137612-sxaiia.jpg?ixlib=rb-1.1.0&q=45&auto=format&w=754&fit=clip" alt="Elderly lady using laptop at home" /></a> Older people will still have to navigate a complex system and make market choices largely on their own. <a href="https://www.shutterstock.com/image-photo/asian-old-woman-using-computer-69690976" class="source">Shutterstock</a> There are currently almost <a href="https://gen-agedcaredata.gov.au/www_aihwgen/media/Home_care_report/Home-Care-Data-Report-1st-Qtr-2021-22.pdf">75,000 waiting for the home care support they need</a>, with some having waited up to nine months. We calculate that up to 15% more home-care places than planned could be needed just to clear the waiting list. We call on the federal government to keep waiting times to 30 days or less. The government’s budget package does include additional support to help older people navigate their way through the home-care system. But assessment, care finding, and care coordination will continue to be fragmented. In the main, older people will still have to navigate a complex system and make market choices on their own. <h2>We need to go local to provide the best support</h2> Australia needs a new home care model – one that provides much more personalised support to help older people get the services they need and that manages local service systems on their behalf. It’s difficult to see this being done without establishing effective regional aged-care offices. These offices need to provide a one-stop shop for older people. Yet they also need to have the authority and responsibility to develop and manage local services to make sure older people can get what they need. The federal government is aware of this problem, but its response is tepid – <a href="https://www.health.gov.au/sites/default/files/documents/2021/05/governance-pillar-5-of-the-royal-commission-response-strengthening-regional-stewardship-of-aged-care.pdf">a trial</a> of small, regional offices of up to ten people to plan, monitor and solve problems. But those regional offices have no responsibility for supporting older people, and no authority to manage service providers on their behalf. We recommend the federal government establish a network of regional aged-care offices across Australia to plan and develop services, hold funds, pay providers, and administer service agreements for individual older people who need care. These offices should include assessment teams and care finders, to help people who are trying to navigate the home-care system. Good quality home care depends on a well-qualified, secure and valued workforce. Again, the federal government is aware of this problem and has introduced a limited set of workforce reforms. But it has not yet agreed to support improved pay and conditions, minimum qualification standards or a full registration scheme for personal-care workers. The government should develop and implement a revitalised workforce plan for aged care as part of the <a href="https://www.health.gov.au/initiatives-and-programs/aged-care-reforms/aged-care-legislative-reform">new Aged Care Act</a>. Personal-care workers should be registered and hold suitable minimum qualifications. The government should also make it clear it will fund the outcomes of the <a href="https://www.fwc.gov.au/cases-decisions-orders/major-cases/work-value-case-aged-care-industry">Fair Work Commission</a> review of fair pay and conditions for aged-care workers, with a ruling expected next year. As Australia’s population continues to age, many more people with complex needs will need care. The vast majority of them will prefer to be supported at home. Massively expanding home-care services without much stronger market management, and a much more secure workforce, is a risk Australia shouldn’t take.<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important; text-shadow: none !important;" src="https://counter.theconversation.com/content/173326/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/stephen-duckett-10730">Stephen Duckett</a>, Director, Health and Aged Care Program, <a href="https://theconversation.com/institutions/grattan-institute-1168">Grattan Institute</a> and <a href="https://theconversation.com/profiles/hal-swerissen-9722">Hal Swerissen</a>, Emeritus Professor, La Trobe University, and Fellow, Health Program, <a href="https://theconversation.com/institutions/grattan-institute-1168">Grattan Institute</a> This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/older-australians-are-already-bamboozled-by-a-complex-home-care-system-so-why-give-them-more-of-the-same-173326">original article</a>. Image: Shutterstock

Caring

Overhaul of payments system to cover digital wallets, buy now pay later, cryptocurrency

Treasurer Josh Frydenberg will announce on Wednesday a comprehensive reform of regulations governing the payments system, to bring it up to date with innovations such as digital wallets and cryptocurrency. The government says without the changes – the biggest in 25 years – Australians businesses and consumers could increasingly be making transactions in spaces beyond the full reach of Australian law, where rules were determined by foreign governments and multinationals. It points out that in three decades payment methods have gone from cash to cheques, cheques to credit cards, credit cards to debit cards and now to “tap and go” via digital wallets on phones or watches. Around a decade ago, cryptocurrency was a concept. Currently, there are more than 220 million participants in the worldwide crypto market, including many in Australia. The planned reforms will centralise oversight of the payment system by ensuring government plays a greater leadership role. The treasurer will be given more power to intervene in certain circumstances. Consumer protection will be strengthened, and more competition and innovation will be promoted. The reform program will be in two phases. There will be consultations in the first half of next year on those that are most urgent and easy to implement. Consultations on the rest will be done by the end of the year. The government says the present one-size-fits-all licensing framework for payment service providers will be replaced graduated, risk-based regulatory requirements. There will be consideration of the feasibility of a retail central bank digital currency, and an examination of “de-banking” (where a bank declines to offer a service to a business or individual). Frydenberg says the comprehensive payments and crypto asset reform program would “firmly place Australia among a handful of lead countries in the world. "It is how we will capitalise on the opportunity for Australia to lead the world in this emerging and fast-growing area which has almost endless potential applications across the economy,” he says. “For businesses, these reforms will address the ambiguity that can exist about the regulatory and tax treatment of crypto assets and new payment methods. "In doing so, it will drive even more consumer interest, facilitate even more new entrants and enable even more innovation to take place. "For consumers, these changes will establish a regulatory framework to underpin their growing use of crypto assets and clarify the treatment of new payment methods.”<img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important; text-shadow: none !important;" src="https://counter.theconversation.com/content/173331/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /> <a href="https://theconversation.com/profiles/michelle-grattan-20316">Michelle Grattan</a>, Professorial Fellow, <a href="https://theconversation.com/institutions/university-of-canberra-865">University of Canberra</a> This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/overhaul-of-payments-system-to-cover-digital-wallets-buy-now-pay-later-cryptocurrency-173331">original article</a>. Image: Shutterstock

Retirement Income

AI-system promises better art reproductions – but not yet

<div> <div class="copy"> A team from the Computer Science and Artificial Intelligence Laboratory at the Massachusetts Institute of Technology in the US is developing a new, deep learning-assisted system to reproduce art with a 3D printer to make more accurate, convincing reproductions. The system combines a process known as halftoning, which uses little dots of ink, and a layering technique that has 10 different colours, rather than the usual cyan, magenta, yellow, and black of 2D printers. This keeps the ink from blotting, which happens when too much is deposited on the printing surface, and it allows the printer to produce a wider range of tones. The technique, combined with a “deep learning model to predict the optimal stack of different inks”, results in “unprecedented spectral accuracy”, the team writes in a new paper, being presented this month at a <a rel="noopener" href="https://sa2018.siggraph.org/en/" target="_blank">computer graphics conference in Tokyo</a>. “If you just reproduce the colour of a painting as it looks in the gallery, it might look different in your home,” says Changil Kim, one of the paper’s authors. “Our system works under any lighting condition, which shows a far greater colour reproduction capability than almost any other previous work.”{%recommended 6743%} The researchers they hope the project will eventually make art more available, since “our reliance on museums to exhibit original paintings and sculpture inherently limits access and leaves those precious originals vulnerable to deterioration and damage”. “The value of fine art has rapidly increased in recent years, so there’s an increased tendency for it to be locked up in warehouses away from the public eye,” notes mechanical engineer Mike Foshey. “We’re building the technology to reverse this trend, and to create inexpensive and accurate reproductions that can be enjoyed by all.” The developers concede that there is still work to be done on the system, which they named RePaint, to truly render a van Gogh simulacrum. For starters, images like Starry Night use a cobalt blue that the ink library isn’t able to “faithfully reproduce”. But paintings – particularly oil paintings – are three-dimensional works. The brush strokes leave ridges and bumps that can reflect light, throwing off the rendering. Right now, the printer reads glossy reflections as white highlights, but the team has plans to incorporate recognition of “the rich spatially-varying gloss and translucency found in many paintings”. The system will learn to use surface reflection, rather than less colour, to reproduce the gloss. One other issue? Those glorious Monet water lilies look more like postage stamps, since the system’s reproductions are only a few centimetres across. The engineers are hoping to bring down the costs and time printing to accommodate larger reproductions. Image credit: Shutterstock  <img id="cosmos-post-tracker" style="opacity: 0; height: 1px!important; width: 1px!important; border: 0!important; position: absolute!important; z-index: -1!important;" src="https://syndication.cosmosmagazine.com/?id=23508&title=AI-system+promises+better+art+reproductions+%E2%80%93+but+not+yet" alt="" width="1" height="1" />  </div> <div id="contributors"> This article was originally published on <a rel="noopener" href="https://cosmosmagazine.com/technology/ai-system-promises-better-art-reproductions-but-not-yet/" target="_blank">cosmosmagazine.com</a> and was written by Samantha Page. </div> </div>

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Overhaul of payments system to cover digital wallets, buy now pay later, cryptocurrency

AI-system promises better art reproductions – but not yet